Figure 1 from Peripheral facial palsy : Grading, Etiology, and Melkersson-Rosenthal Syndrome | Semantic Scholar (2024)

It is demonstrated that surface electromyogram (sEMG) can be used to develop an automated, objective, and quantitative grading system for assessment of facial functions and muscles.

This review will provide the clinicians and researchers a brief overview of the facial grading systems which have been used and assist in the development of a standard system for facial paralysis evaluation.

The proposed method includes preprocessing of images and estimation of level of diseases and measures the distance between the eye brows to infra orbital to evaluate the degree of facial paralysis by using IECM algorithm.

It was concluded that 3-D motion analyses by Qualisys motion capture system can be considered as a reliable method for assessment of Bell's palsy and can detect and characterize a wide range of clinically significant facial functional deficits.

The developed FP grading system provides a detailed quantitative report and has significant advantages over the existing grading scales and is suitable to be used as a clinical tool.

A technique is proposed to evaluate the degree of facial paralysis using well known neural network called Feed Forward Back Propagation Neural Network (FFBPN).

The present research aims to enhance the accuracy and robustness of one of this system's modules: the resting symmetry module by including several modifications to the computation method of the symmetry index (SI) for the eyebrows, eyes and mouth.

The present study describes the implementation of the second module for grading voluntary movements of the Automated Facial Grading (AFG) system, which utilizes the Kinect v2 sensor to detect and capture facial landmarks in real time.

A technique is proposed to evaluate the degree of facial paralysis using Salient Point Selection Algorithm (SPSA) and Degree Evaluation (DE).

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A prospective study in which a well‐defined surgical decompression of the facial nerve was performed in a population of patients with Bell's palsy who exhibit the electrophysiologic features associated with poor outcomes.

Lyme disease seems to be an infrequent cause of facial palsy in patients without constitutional symptoms or additional neurological findings, and peripheral facial palsies is a common disorder with a favourable prognosis.

A survey of the literature showed that no kind of treatment, including prednisone, was able to give a better prognosis and the use ofprednisone raises a big ethical problem because no evidence of its efficacy exists and the euphoric side-effect induces a false feeling of benefit in the patients.

Cerebrospinal fluid is usually normal in idiopathic peripheral facial palsy, and if the CSF is abnormal, a specific cause should be sought.

A 9‐year‐old girl is described who had episodic swelling of the upper lip and complete peripheral facial palsy, associated with herpes and recurrent bacterial infections, and benefited from a 2‐month course of prednisolone 1 mg kg−1 daily.

It is indicated that a small percentage (approximately 8%) of patients with otherwise typical Bell's palsy may harbor additional cranial neuropathies.

Transmastoid facial nerve surgery is not recommended to treat Bell's palsy because no benefits have been identified which outweigh the risks of surgery and outcome in patients treated surgically was no better than can be expected to occur without treatment.

It is concluded that Bell's palsy is an acute benign cranial polyneuritis probably caused by reactivation of the herpes‐simplex virus, and the dysfunction of the motor cranial nerves may represent inflammation and demyelinization rather than ischemic compression.

The results show that any diagnosis of HHV-6 encephalitis or other type of active central nervous system infection should not be made without first excluding chromosomal HHv-6 integration by measuring DNA load in CSF, serum, and/or whole blood.

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